“In any case, UVC is strongly attenuated by chromophores in the upper epidermis (Young, 1997) and UVC-induced DNA damage in the dividing basal layer of human epidermis is not readily detected (Campbell et al, 1993; Chadwick et al, 1995) which may explain why the dose response curve for UVC erythema in human skin is very much less steep than for UVB (Diffey and Farr, 1991). It is unlikely that UVC from artificial sources presents an acute or long-term hazard to human skin. However, UVC is likely to cause acute photokeratitis… UVC exposure is unlikely to cause acute or long-term damage to the skin but can cause severe acute damage to the eye and should not be permitted at all from any tanning device.”
https://www.klaran.com/klaran-university/about-uvc
https://www.sciencedirect.com/science/article/pii/S1466856410001050?via%3Dihub
Research Highlights
►UV-C light causes 2 log reductions in total viable count of melon cubes.
►UV-C light inhibits microbial growth during storage of fresh-cut melon at 6 °C.
The UV spectrum of sunlight is conventionally divided into short wavelength UVC (λ < 280 nm), mid-wavelength UVB (λ 290–320 nm), and long wavelength UVA (λ > 320–400 nm).46 The UVC fraction of sunlight is entirely absorbed by stratospheric oxygen (O2), which subsequently undergoes decomposition and recombination reactions, giving rise to ozone (O3). The resulting O3 molecules can function as a filter and absorb the majority of sunlight UVB.47,48 Thus, the solar UV wavebands that reach the surface of the earth, and as such are of relevance for photocarcinogenesis, are UVA and UVB, which comprise 95% and 5%, respectively, of the terrestrial sunlight UV48,49 (Fig. 1).
© Copyright SaniSentry